<?xml version="1.0" encoding="utf-8" standalone="yes"?><rss version="2.0" xmlns:atom="http://www.w3.org/2005/Atom"><channel><title>"Cardiac/drug effects/*metabolism" | Glembotski Lab</title><link>https://www.glembotskilab.org/tag/cardiac/drug-effects/metabolism/</link><atom:link href="https://www.glembotskilab.org/tag/cardiac/drug-effects/metabolism/index.xml" rel="self" type="application/rss+xml"/><description>"Cardiac/drug effects/*metabolism"</description><generator>Wowchemy (https://wowchemy.com)</generator><language>en-us</language><copyright>© Glembotski Lab</copyright><image><url>https://www.glembotskilab.org/media/icon_hua2ec155b4296a9c9791d015323e16eb5_11927_512x512_fill_lanczos_center_3.png</url><title>"Cardiac/drug effects/*metabolism"</title><link>https://www.glembotskilab.org/tag/cardiac/drug-effects/metabolism/</link></image><item><title>Proteomic analysis of the cardiac myocyte secretome reveals extracellular protective functions for the ER stress response.</title><link>https://www.glembotskilab.org/publication/blackwood-2020-j-mol-cell-cardiol-143-132/</link><pubDate>Mon, 01 Jun 2020 00:00:00 +0000</pubDate><guid>https://www.glembotskilab.org/publication/blackwood-2020-j-mol-cell-cardiol-143-132/</guid><description/></item><item><title>Junctophilin-2 gene therapy rescues heart failure by normalizing RyR2-mediated Ca(2+) release.</title><link>https://www.glembotskilab.org/publication/reynolds-2016-int-j-cardiol-225-371/</link><pubDate>Thu, 01 Dec 2016 00:00:00 +0000</pubDate><guid>https://www.glembotskilab.org/publication/reynolds-2016-int-j-cardiol-225-371/</guid><description/></item><item><title>LPS-induced autophagy is mediated by oxidative signaling in cardiomyocytes and is associated with cytoprotection.</title><link>https://www.glembotskilab.org/publication/yuan-2009-am-j-physiol-heart-circ-physiol-296-h-470/</link><pubDate>Sun, 01 Feb 2009 00:00:00 +0000</pubDate><guid>https://www.glembotskilab.org/publication/yuan-2009-am-j-physiol-heart-circ-physiol-296-h-470/</guid><description/></item></channel></rss>